aea cayman chemical Search Results


aea  (Larodan)
93
Larodan aea
Aea, supplied by Larodan, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/aea+cayman+chemical/2-Monoarachidonin/pm21749725-90-20-45
Average 93 stars, based on 1 article reviews
aea - by Bioz Stars, 2026-10
93/100 stars
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aea  (Tocris)
93
Tocris aea
Fig. 11 C6 cell proliferation at 48 h. a Concentration-dependent effect <t>of</t> <t>ACEA,</t> <t>AEA</t> and AA- 5-HT (10−8−10−6 M) in the presence or absence of SR141716A (10−7−10−6 M) on MPTP (100 μM)-induced cell proliferation. Effect of ACEA and SR141716A (10−6 M) given alone on C6 cell proliferation. Maximal concentration of ACEA, AEA and AA-5-HT (10−6 M) was used in the pres- ence of CB1 antagonist. b Effects of SR141716A and SR144528 (10−6 M) on MPTP (100 μM)-induced cell prolifer- ation. c Effect of VDM-11 (10−7−10−6 M) given 30 min before MPTP or 12 h after MPTP. Maximal concentration of VDM-11 (10−6 M) was used in the presence of CB1 antago- nist (10−7−10−6 M). Each bar shows the mean±SEM of n=4 experiments. ***P<0.001 vs control; °P<0.05, °°P<0.01, °°°P<0.001 vs MPTP; §P<0.05, §§P<0.01, §§§P<0.001 vs MPTP; #P<0.05, ##P<0.01, ###P<0.001 vs MPTP. ##P<0.05 and ###P<0.001 vs ACEA @@P<0.05 and @@@P<0.001 vs AEA; §§P<0.05 and §§§P<0.001 vs AA-5-HT
Aea, supplied by Tocris, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/aea+cayman+chemical/Anandamide/pm17639288-40-24-30
Average 93 stars, based on 1 article reviews
aea - by Bioz Stars, 2026-10
93/100 stars
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pixantrone maleate  (Selleck Chemicals)
93
Selleck Chemicals pixantrone maleate
Fig. 11 C6 cell proliferation at 48 h. a Concentration-dependent effect <t>of</t> <t>ACEA,</t> <t>AEA</t> and AA- 5-HT (10−8−10−6 M) in the presence or absence of SR141716A (10−7−10−6 M) on MPTP (100 μM)-induced cell proliferation. Effect of ACEA and SR141716A (10−6 M) given alone on C6 cell proliferation. Maximal concentration of ACEA, AEA and AA-5-HT (10−6 M) was used in the pres- ence of CB1 antagonist. b Effects of SR141716A and SR144528 (10−6 M) on MPTP (100 μM)-induced cell prolifer- ation. c Effect of VDM-11 (10−7−10−6 M) given 30 min before MPTP or 12 h after MPTP. Maximal concentration of VDM-11 (10−6 M) was used in the presence of CB1 antago- nist (10−7−10−6 M). Each bar shows the mean±SEM of n=4 experiments. ***P<0.001 vs control; °P<0.05, °°P<0.01, °°°P<0.001 vs MPTP; §P<0.05, §§P<0.01, §§§P<0.001 vs MPTP; #P<0.05, ##P<0.01, ###P<0.001 vs MPTP. ##P<0.05 and ###P<0.001 vs ACEA @@P<0.05 and @@@P<0.001 vs AEA; §§P<0.05 and §§§P<0.001 vs AA-5-HT
Pixantrone Maleate, supplied by Selleck Chemicals, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/aea+cayman+chemical/Pixantrone+Maleate/bio_rxiv__2021__10__07__463234-90-10-25
Average 93 stars, based on 1 article reviews
pixantrone maleate - by Bioz Stars, 2026-10
93/100 stars
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20 hete ethanolamide  (Cayman Chemical)
N/A
Arachidonoyl ethanolamide AEA is an endogenous lipid neurotransmitter with cannabinergic activity binding both the central cannabinoid CB and peripheral cannabinoid CB receptors Fatty acid amide hydrolase FAAH is the enzyme responsible for the hydrolysis and
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o arachidonoyl ethanolamine  (Cayman Chemical)
N/A
Arachidonoyl ethanolamide AEA was the first endogenous cannabinoid to be isolated and characterized as an agonist acting on the same receptors CB and CB as tetrahydrocannabinols THC Since that time a number of related endocannabinoids
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arachidonoyl ethanolamide maxspec standard  (Cayman Chemical)
N/A
Arachidonoyl ethanolamide AEA is the ethanolamine amide of arachidonic acid Item No 90010 first isolated from porcine brain AEA is an endogenous cannabinoid neurotransmitter that binds to both central cannabinoid CB and peripheral cannabinoid CB
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2 methyl arachidonoyl 2 fluoroethylamide  (Cayman Chemical)
N/A
2 Methyl arachidonoyl 2 fluoroethylamide 2 Methyl 2 fluoro AEA is an analog of anandamide AEA in which the alcohol of the ethanolamide group has been removed and replaced with a fluorine atom This substitution
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arachidonoyl ethanolamide  (Cayman Chemical)
N/A
Arachidonoyl ethanolamide AEA is the ethanolamine amide of arachidonic acid originally isolated from porcine brain AEA is an endogenous cannabinoid neurotransmitter that binds to both cannabinoid 1 CB and CB receptors AEA has K values
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arachidonoyl ethanolamide d4  (Cayman Chemical)
N/A
Arachidonoyl ethanolamide d AEA d contains four deuterium atoms at the hydroxyethyl 1 1 2 and 2 positions It is intended for use as an internal standard for the quantification of AEA by GC or
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arachidonoyl 2 chloroethylamide  (Cayman Chemical)
N/A
Arachidonoyl 2 chloroethylamide ACEA is a potent and selective cannabinoid CB receptor 1 agonist with K values of 1 4 and 2 000 nM for CB and CB receptors respectively In whole animal experiments ACEA
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arachidonoyl 2 fluoroethylamide  (Cayman Chemical)
N/A
Arachidonoyl 2 fluoroethylamide 2 fluoro AEA is an analog of anandamide in which the alcohol of the ethanolamide group has been removed and replaced with a fluorine atom This substitution adds considerably increased binding affinity
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oxy arachidonoyl ethanolamide  (Cayman Chemical)
N/A
AEA acts as an endogenous mimic of Δ THC the psychotropic component of marijuana oxy AEA is the O alkyl N acyl oxyhomologue of AEA oxy AEA is selective for the peripheral cannabinoid CB receptor
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Image Search Results


Fig. 11 C6 cell proliferation at 48 h. a Concentration-dependent effect of ACEA, AEA and AA- 5-HT (10−8−10−6 M) in the presence or absence of SR141716A (10−7−10−6 M) on MPTP (100 μM)-induced cell proliferation. Effect of ACEA and SR141716A (10−6 M) given alone on C6 cell proliferation. Maximal concentration of ACEA, AEA and AA-5-HT (10−6 M) was used in the pres- ence of CB1 antagonist. b Effects of SR141716A and SR144528 (10−6 M) on MPTP (100 μM)-induced cell prolifer- ation. c Effect of VDM-11 (10−7−10−6 M) given 30 min before MPTP or 12 h after MPTP. Maximal concentration of VDM-11 (10−6 M) was used in the presence of CB1 antago- nist (10−7−10−6 M). Each bar shows the mean±SEM of n=4 experiments. ***P<0.001 vs control; °P<0.05, °°P<0.01, °°°P<0.001 vs MPTP; §P<0.05, §§P<0.01, §§§P<0.001 vs MPTP; #P<0.05, ##P<0.01, ###P<0.001 vs MPTP. ##P<0.05 and ###P<0.001 vs ACEA @@P<0.05 and @@@P<0.001 vs AEA; §§P<0.05 and §§§P<0.001 vs AA-5-HT

Journal: Journal of molecular medicine (Berlin, Germany)

Article Title: Cannabinoid CB1 receptor stimulation affords neuroprotection in MPTP-induced neurotoxicity by attenuating S100B up-regulation in vitro.

doi: 10.1007/s00109-007-0233-y

Figure Lengend Snippet: Fig. 11 C6 cell proliferation at 48 h. a Concentration-dependent effect of ACEA, AEA and AA- 5-HT (10−8−10−6 M) in the presence or absence of SR141716A (10−7−10−6 M) on MPTP (100 μM)-induced cell proliferation. Effect of ACEA and SR141716A (10−6 M) given alone on C6 cell proliferation. Maximal concentration of ACEA, AEA and AA-5-HT (10−6 M) was used in the pres- ence of CB1 antagonist. b Effects of SR141716A and SR144528 (10−6 M) on MPTP (100 μM)-induced cell prolifer- ation. c Effect of VDM-11 (10−7−10−6 M) given 30 min before MPTP or 12 h after MPTP. Maximal concentration of VDM-11 (10−6 M) was used in the presence of CB1 antago- nist (10−7−10−6 M). Each bar shows the mean±SEM of n=4 experiments. ***P<0.001 vs control; °P<0.05, °°P<0.01, °°°P<0.001 vs MPTP; §P<0.05, §§P<0.01, §§§P<0.001 vs MPTP; #P<0.05, ##P<0.01, ###P<0.001 vs MPTP. ##P<0.05 and ###P<0.001 vs ACEA @@P<0.05 and @@@P<0.001 vs AEA; §§P<0.05 and §§§P<0.001 vs AA-5-HT

Article Snippet: Anti-S100B antibody was from AbCam (Cambridge, UK), anti-CB1 antibody was from Affinity BioReagent (Golden, CO) and anti-CB2 antibody was from Cayman (Ann Arbor, MI); AEA, arachidonyl-2-chloroethylamide (ACEA), JWH015 were from Tocris (Cookson, Bristol, UK), SR141716A and SR144528 were a gift from Dr. Madaleine Mosse (SANOFI-Reserche, Montpellier, France) and VDM-11 and arachydonoylserotonin (AA-5-HT) were synthesized in the laboratory of prof. Vincenzo Di Marzo (CNR-Pozzuoli, Italy).

Techniques: Concentration Assay, Control

Fig. 12 Effect of ACEA, AEA, AA-5-HT and SR141716A giv- en to MPTP-treated C6 cell on co-cultured PC12 cell viability. a Concentration-dependent effects of ACEA, AEA and AA- 5-HT in the presence or absence of SR141716A. Maximal con- centrations of ACEA, AEA and AA-5-HT (10−6 M) were used in the presence of the CB1 antago- nist. b Concentration-dependent effect of SR141716A. Each bar shows the mean±SEM of n=3 experiments. °°°P<0.001 vs control; *P<0.05, **P<0.01, ***P<0.001 vs MPTP

Journal: Journal of molecular medicine (Berlin, Germany)

Article Title: Cannabinoid CB1 receptor stimulation affords neuroprotection in MPTP-induced neurotoxicity by attenuating S100B up-regulation in vitro.

doi: 10.1007/s00109-007-0233-y

Figure Lengend Snippet: Fig. 12 Effect of ACEA, AEA, AA-5-HT and SR141716A giv- en to MPTP-treated C6 cell on co-cultured PC12 cell viability. a Concentration-dependent effects of ACEA, AEA and AA- 5-HT in the presence or absence of SR141716A. Maximal con- centrations of ACEA, AEA and AA-5-HT (10−6 M) were used in the presence of the CB1 antago- nist. b Concentration-dependent effect of SR141716A. Each bar shows the mean±SEM of n=3 experiments. °°°P<0.001 vs control; *P<0.05, **P<0.01, ***P<0.001 vs MPTP

Article Snippet: Anti-S100B antibody was from AbCam (Cambridge, UK), anti-CB1 antibody was from Affinity BioReagent (Golden, CO) and anti-CB2 antibody was from Cayman (Ann Arbor, MI); AEA, arachidonyl-2-chloroethylamide (ACEA), JWH015 were from Tocris (Cookson, Bristol, UK), SR141716A and SR144528 were a gift from Dr. Madaleine Mosse (SANOFI-Reserche, Montpellier, France) and VDM-11 and arachydonoylserotonin (AA-5-HT) were synthesized in the laboratory of prof. Vincenzo Di Marzo (CNR-Pozzuoli, Italy).

Techniques: Cell Culture, Concentration Assay, Control